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Yann Quide


Postdoctoral Research Fellow, UNSW School of Psychiatry Affiliated Scientist, NeuRA

02 9399 1866

I am an early-career postdoctoral researcher in the School of Psychiatry UNSW Sydney and an affiliated scientist at Neuroscience Research Australia (NeuRA). I received a PhD scholarship from the French Ministry of Higher Education and Research to complete my doctoral studies at the University of Tours (France). I subsequently joined the Research Unit for Schizophrenia Epidemiology at UNSW and the Stress-related Psychopathology research stream at NeuRA.

I have a particular interest in identifying the neurobiological underpinnings of stress and trauma in relation to psychiatric conditions. My present research interests are two-fold:

  • First, I am interested in the investigation of the long-term neurobiological effects of childhood trauma exposure on the risk of developing schizophrenia and bipolar disorder. In particular, I study the effects of childhood trauma exposure interacting with genetics, epigenetics (methylation), neuroendocrine (cortisol) and inflammatory (cytokines) markers to affect brain structure and function, and neuropsychological performance.
  • Second, in collaboration with the INSERM Unit 1253 at the University of Tours (France), I am interested in the identification of the early neurobiological (neuroendocrine, neuroimaging) and neuropsychological changes occurring in the aftermath of sexual assault in females, and how these lead to the development of posttraumatic stress disorder (PTSD).
Projects Yann Quide is currently involved with


Childhood trauma and inflammatory markers

This project examines immune and stress response markers in association with epigenetic markers and brain structure/function in psychotic disorders.


Childhood Trauma and Inflammatory Markers

Epigenetic and childhood trauma in psychotic and mood disorder

This project examines epigenetic (methylation) markers of childhood trauma in schizophrenia and bipolar disorder patients.


Epigenetics and childhood trauma

Imaging Genetics in Psychosis Study

The Imaging-Genetics in Psychosis study aims to determine common stress-related pathology among schizophrenia and bipolar disorder patients, in association with shared genetic vulnerabilities, cognitive deficits, and brain phenotypes.


Imaging Genetics in Psychosis Study









DR KRISTIN LAURENS Senior Research Scientist


KIMBERLIE DEAN Principle Research Scientist

FELICITY HARRIS Research Officer

PROF VAUGHAN CARR Senior Principle Research Scientist


Neurocognitive, emotional and neuroendocrine correlates of exposure to sexual assault in women.

Quidé Y, Cléry H, Andersson F, Descriaud C, Saint-Martin P, Barantin L, Gissot V, Carrey Le Bas MP, Osterreicher S, Dufour-Rainfray D, Brizard B, Ogielska M, El-Hage W

Dysfunctions in the dorsal anterior cingulate cortex and the cerebellum may represent early functional brain modifications that alter higher cognitive processes when emotional material is involved.

Common variation in ZNF804A (rs1344706) is not associated with brain morphometry in schizophrenia or healthy participants.

Quidé Y, Matosin N, Atkins JR, Fitzsimmons C, Cairns MJ, Carr VJ, , Green MJ

Contrary to some – but not all – previous findings, this study of a large sample of schizophrenia cases and healthy controls reveals no evidence for association between grey matter alterations and variation in rs1344706 (ZNF804A). Differences in sample sizes and ethnicities may account for discrepant findings between the present and previous studies.

Action, observation or imitation of virtual hand movement affect differently regions of the mirror neuron system and the default mode network.

Brihmat N, Tarri M, Quidé Y, Anglio K, Pavard B, Castel-Lacanal E, Gasq D, De Boissezon X, Marque P, Loubinoux I

Virtual reality (VR)-based paradigms use visual stimuli that can modulate visuo-motor networks leading to the stimulation of brain circuits. The aims of this study were to compare the changes in blood-oxygenation level dependent (BOLD) signal when watching and imitating moving real (RH) and virtual hands (VH) in 11 healthy participants (HP). No differences were found between the observation of RH or VH making this VR-based experiment a promising tool for rehabilitation protocols. VH-imitation involved more the ventral premotor cortex (vPMC) as part of the mirror neuron system (MNS) compared to execution and VH-observation conditions. The dorsal-anterior Precuneus (da-Pcu) as part of the Precuneus/posterior Cingulate Cortex (Pcu/pCC) complex, a key node of the Default Mode Network (DMN), was also less deactivated and therefore more involved. These results may reflect the dual visuo-motor roles for the vPMC and the implication of the da-Pcu in the reallocation of attentional and neural resources for bimodal task management. The ventral Pcu/pCC was deactivated regardless of the condition confirming its role in self-reference processes. Imitation of VH stimuli can then modulate the activation of specific areas including those belonging to the MNS and the DMN.

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